Atopic Dermatitis (cont.)
Gary W. Cole, MD, FAAD
Dr. Cole is board certified in dermatology. He obtained his BA degree in bacteriology, his MA degree in microbiology, and his MD at the University of California, Los Angeles. He trained in dermatology at the University of Oregon, where he completed his residency.
William C. Shiel Jr., MD, FACP, FACR
Dr. Shiel received a Bachelor of Science degree with honors from the University of Notre Dame. There he was involved in research in radiation biology and received the Huisking Scholarship. After graduating from St. Louis University School of Medicine, he completed his Internal Medicine residency and Rheumatology fellowship at the University of California, Irvine. He is board-certified in Internal Medicine and Rheumatology.
In this Article
- Atopic dermatitis facts
- What is atopic dermatitis?
- What is the difference between atopic dermatitis and eczema?
- How common is atopic dermatitis?
- What causes atopic dermatitis?
- Is atopic dermatitis contagious?
- What are atopic dermatitis symptoms and signs?
- Can atopic dermatitis affect the face?
- What are the stages of atopic dermatitis?
- How do physicians diagnose atopic dermatitis?
- What factors can aggravate atopic dermatitis?
- What are skin irritants in patients with atopic dermatitis?
- What are allergens?
- What are aeroallergens?
- What is the treatment for atopic dermatitis?
- What is the prognosis of atopic dermatitis?
- Find a local Dermatologist in your town
Can atopic dermatitis affect the face?
Yes. Atopic dermatitis may affect the skin around the eyes, the eyelids, the eyebrows, and lashes. Scratching and rubbing the eye area can cause the skin to change in appearance. Some people with atopic dermatitis develop an extra fold of skin under their eyes, called an atopic pleat or Dennie-Morgan fold. Other people may have hyperpigmented eyelids, meaning that the skin on their eyelids darkens from the inflammation or hay fever (allergic shiners). Patchy eyebrows and eyelashes may also result from scratching or rubbing.
The face is very commonly affected in babies, who may drool excessively, and become irritated from skin contact with their flowing saliva.
The skin of a person with atopic dermatitis loses too much moisture from the epidermal layer. Some patients with atopic dermatitis lack a protein called fillagrin that is important in retaining moisture. This defective genetic trait allows the skin to become very dry, which reduces its protective abilities. In addition, the skin is very susceptible to recurring disorders, such as staphylococcal and streptococcal bacterial skin infections, warts, herpes simplex, and molluscum contagiosum (which is caused by a virus).
Skin features of atopic dermatitis
- Lichenification: thick, leathery skin resulting from constant scratching and rubbing
- Lichen simplex: refers to a thickened patch of raised skin that results from repeat rubbing and scratching of the same skin area
- Papules: small, raised bumps that may open when scratched, becoming crusty and infected
- Ichthyosis: dry, rectangular scales on the skin, commonly on the lower legs and shins
- Keratosis pilaris: small, rough bumps, generally on the face, upper arms, and thighs. These are also described as gooseflesh or chicken skin and may have a small coiled hair under each bump.
- Hyper linear palms: increased number of skin creases on the palms
- Urticaria: hives (red, raised bumps), often after exposure to an allergen, at the beginning of flares, or after exercise or a hot bath
- Cheilitis: inflammation of the skin on and around the lips
- Atopic pleat (Dennie-Morgan fold): an extra fold of skin that develops under the eye
- Dark circles under the eyes: may result from allergies and atopy
- Hyperpigmented eyelids: eyelids that have become darker in color from inflammation or hay fever
- Prurigo nodules also called "picker's warts" are not really warts at all. These are small thickened bumps of skin caused by repeated picking of the same skin site.
|Picture of nummular eczema on the leg|
Find out what women really need.